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Troponin I degradation and myocardial stunning.

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Calpain inhibition preserves talin and attenuates right heart failure in acute pulmonary hypertension. Calpaindependent degradation of troponin I mutants found in familial hypertrophic cardiomyopathy: Molecular and Cellular Biochemistry.

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A novel phosphorylation site, Serinein the C-terminus of cardiac troponin I regulates calcium sensitivity and susceptibility to calpain-induced proteolysis. Effect of hypertrophic and dilated cardiomyopathies associated mutations in troponin I on cardiac thin filament dynamics.

Hypertrophic cardiomyopathy. Calcineurin signalling mechanisms in myocardial hypertrophy.


Hypertrophic Cardiomyopathy. Calpain activation impairs neuromuscular transmission canadian journal of diabetes 2021 a mouse model of the slow-channel myasthenic syndrome. A switch that lowers the beta AR: insights from a troponin I mutation linked to hypertrophic cardiomyopathy. Molecular determinants of myofibrillar contractile function in the heart. Alterations in myofilament function contribute to left ventricular dysfunction in pigs early after myocardial infarction.


Calpainsensitive myofibrillar proteins of the human myocardium. Advances in point-of-care testing for cardiovascular diseases.

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Bromine inhalation mimics ischemia-reperfusion cardiomyocyte injury and calpain activation in rats. Acute ischemia and Reperfusion syndrome. Transcriptional analysis of caveolae - related transcripts in the ischaemic - intolerant ageing mouse heart.

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Emerging importance of oxidative stress in regulating striated muscle az alkoholizmus kezelés cukorbetegség. Degradation systems in heart failure. Troponin I phosphorylation in human myocardium in health and disease.

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Cardiac troponin in ischemic cardiomyocytes: Intracellular decrease before onset of cell death. Atrial calpains: Mediators of atrialmyopathies in atrial fibrillation. Muscle wasting in cancer.

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Intracellular matrix remodeling and cardiac function in ischemia-reperfusion injury. Cooperation between proteolytic canadian journal of diabetes 2021 in cardiomyocyte recycling. Calcium fluxes and homeostasis. Analysis of myotilin turnover provides mechanistic insight into the role of myotilinopathy-causing mutations. Over-expression of calpastatin inhibits calpain activation and attenuates myocardial dysfunction during endotoxaemia.

Bővebben: A cukorbetegség biokémiai-élettani háttere A tápcsatorna a táplálékkal felvett összetett szénhidrátokat glükózra monoszacharid — köznapi nevén szőlőcukorra — bontja. Ez azt jelenti, hogy minden emészthető szénhidrát hasonló hatású a szervezetre, bár a lebontás sebessége fontos tényező. A glükóz a bélből felszívódva a vérbe kerül, és ezúton a test minden részére eljut.

Caspase and calpain activation both contribute to sepsis-induced diaphragmatic weakness. Selective vacuolar degeneration in dystrophin-deficient canine Purkinje fibers despite preservation of dystrophin-associated proteins with overexpression of Dp Myofilament proteins: From cardiac disorders to proteomic changes. Calpain mediates cardiac troponin degradation and contractile dysfunction in atrial fibrillation.

Calpain inhibition attenuates right ventricular contractile dysfunction after acute pressure overload. Calcium cycling and signaling in cardiac myocytes. Progression of heart failure was suppressed by inhibition of apoptosis signal-regulating kinase 1 via transcoronary gene transfer.

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Insulin signaling regulates cardiac titin properties in heart development and diabetic cardiomyopathy. Role of oxidative stress in ischemia-reperfusion-induced alterations in myofibrillar ATPase activities and gene expression in the heart. Thyroid hormone analog, diiodothyropropionic acid DITPAexerts beneficial effects on chamber and cellular remodeling in cardiomyopathic hamsters. Cardiovascular and renal manifestations of hyperthyroidism. Antidiabetic potential of oleanolic acid from Ligustrum lucidum Ait.

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Cardiovascular autophagy Concepts, controversies, and perspectives. Reversal of subcellular remodelling by losartan in heart failure due to myocardial infarction.

Overexpression of apolipoprotein B attenuates pathologic cardiac remodeling and hypertrophy in response to catecholamines and after myocardial infarction in mice.

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Subcellular remodelling may induce cardiac dysfunction in congestive heart failure. Impact of maternal undernutrition on diabetes and cardiovascular disease risk in adult offspring. Prevention of heart failure - A scientific statement from the American Heart Association councils on epidemiology and prevention, clinical cardiology, cardiovascular nursing, and high blood pressure research; Quality of care and outcomes research interdisciplinary working group; and functional genomics and translational biology interdisciplinary working group.

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Markers of oxidative damage in chronic heart failure: role in disease progression. Ethanol extract from Epimedium brevicornum attenuates left ventricular dysfunction and cardiac remodeling through down-regulating matrix metalloproteinase-2 and -9 activity and myocardial apoptosis in rats with congestive heart failure. Residual stress ischaemia is associated with blood markers of myocardial structural remodelling. Early imaging in heart failure: Exploring novel molecular targets.

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